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Nowadays there are a variety of breast augmentation methods and each one tries to guarantee the perfect results. Every method tempts with this promise. Let’s review some popular breast augmentation ways… Breast augmentation surgery is one of the most popular cosmetic procedures. Breast augmentation, technically is known as augmentation mammaplasty, is a plastic surgery, which helps to augment the size and shape of a woman’s breast for a number of reasons: - to enhance breast of woman, who fells her breast is small; - to correct a contraction in breast volume after pregnancy; - to balance a difference in breast size. There are several types of breast implants that you can choose depending on you finance. Breast augmentation is believed in immediate and long-lasting results. Nonsurgical breast augmentation methods contain a wide variety of options: herbal remedies, pills, creams, and other devices. Many women can not spend much money on surgical breast augmentation, so they choose nonsurgical one, because of some reasons: it’s not expensive, has such advantages as lower risk of side effects, natural ingredients, and no convalesce period. The most popular alternative method to surgical breast enlargement is breast enhancement pills (also known as bust pills). The phytoestrogens (the material that contains pills) stimulate body to produce new breast tissue growth. It’s written that woman will see the results after 6-8 weeks, but they may vary due to person body chemistry. Also if you want to reach better results you have to follow all recommendations and take additional pills during 90 days after you get the desired size of your breast. Many internet resources present those pills as reliable method that enhance your breast fast and without any surgical procedure. Another method is breast enhancement cream. This cream serves much the same purpose as pills; claims made by the manufacturers of breast enhancement cream products state that individuals using the creams can increase their breast by several cup sizes. Most breast enhancement cream ingredients are identical to those of pills and other supplements, but it does not contain as great concentration of active ingredients as the pills. Breast enhancement cream can help women achieve fuller breasts with firmer tissue. It can be used alone or in combination with breast enhancement pills. Brand-new breast augmentation method is BRAVA system. “BRAVA system is like a bra with two plastic domes that are connected to a suction device, that’s why most patients quickly get used to sleeping in it – because for women it is natural to wear a bra.” You must wear it for 10 hour per a day for 10 weeks. The results will be seen after several months. The main difference between surgical and nonsurgical breast augmentation is the percentage of efficiency. Surgical breast augmentation will bring you immediate and long-lasting result, while nonsurgical devices require much time and nobody guarantees fast and permanent results. Use of this article is permitted as long as there are clickable links back to us at: Breast augmentation and all credit is given to the author. manual penis enlagement health pro solution best enhancement exercise penis best enhancement exercise penis penis elargement natural penis enhancement exercise penis enlargement result penis enargement stretcher

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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. best penis elargement free penis elargement tip penis enargement surgery picture truth about penis enlarement buy pennis enlargement pills pnis enlargement before and after photo vimax penis enlargement before and after photo enlagement manhattan penis surgeon cheap penis enhancement

Maureen Dowd was on Imus the other morning plugging her new book, “Are Men Necessary”; a book I plan to buy so I can get some slightly demented insight into the mind of a troubled woman. During the interview, Imus and his sidekick Charles challenged Ms. Dowd about a female perception she had just suggested that all heterosexual men froth at the mouth at the mere mention of a trip to a strip club or the possibility of a cat fight or the chance two women might lock in lesbian love making. Imus proclaimed that he, even amidst the weakness of lowly cocaine induced comas and vodka fed stupors, never stepped inside a topless joint. Charles nodded his head in brotherhood like the bobble-head doll he is sometimes. Their point being, not all men are beasts; that some have evolved above such shameful sexual servitude. A couple of things. First, Imus and Charles are probably lying through their coffee stained teeth about visiting strip clubs. Second, I have frequented such establishments years ago. I eventually concluded that go-go bars are places where prematurely balding, man-boobed, middle aged business men hire enterprising young shapely women, forming a convenient unholy alliance of distrust to tap into the cash cow created when injured fragile male egos are deceived by alcohol induced sexual fantasy. All the females need to do is squirm provocatively while whispering real sweet nothings into customers’ hair filled ears. And if carried out correctly, the dollars shoot out of the slobbering stooges like ATMs in gleeful male orgasm. Make no mistake about it; the dancer is always in control of the patron. And when she is not, she moves on to the next penis clad cash machine. The only cost to her is to turnover some obscene percentage of the take to her sleazy male boss. It’s a business after all, and business is still a male dominated endeavor. Third, if one has ever listened to Imus for more than an hour, one knows he and his cronies takes delight in sexually stereotyping and demeaning women. This idea that Charles and he are better than that is all part of the act. For instance, a few minutes further into the same interview, Imus commented on the “balls” it took for Maureen Dowd to write a particular op-ed piece about Judith Miller—a remark that she quickly and graciously accepted with a simple and sweet, “thank you”. Although I haven’t checked, I’m going to go out on a limb and say that Ms. Dowd does not have testicles. So why was she so quick to acknowledge and accept what I’m guessing she felt was a compliment? I’m pretty sure that bravery, fearlessness, strength, and conviction—all nice attributes to have when kept in check by common sense—are not gender assigned. And I’m positive they are not a function of male genitalia. I’m equally convinced that reluctance, fearfulness, and weakness do not require one to have a vagina. It’s one thing, a very feeble thing at that, for Imus and his crew or even Jon Stewart and Al Franken for that matter—all professed non-chauvinists—to use male-centric language in an “equal opportunity” way; misguided into believing that somehow they are treating women and men equally. It is another thing though for Maureen Dowd to acknowledge and welcome her inclusion into the club. She could have simply said, “Imus are you suggesting that I have to be a man to be tough?” I am sure if asked Maureen Dowd would say without hesitation that she is a feminist or at least a proponent of feminist beliefs. Why then did she let Imus off the hook and indulge in the myth? Like many things about feminists, I don’t get it. They can be their own worst enemy from time to time—just like Democrats when they run a national campaign. Here is another example of something I don’t get. Why do some corporate feminists find short tight skirts, plunging necklines and push-up bras to be the business suit of choice? I suppose they might argue, just as strippers might, that they are simply using their power over men to get what they want. And on some level I understand that argument: play into the male need to be the sexual alpha dog as long as the targeted objective is personal gain. This attitude however strikes me as feeding the very stereotyping and sexism women want to end, which leads me into a short discussion of another dilemma I have with feminism. Within the last few years, I have been introduced to the forefront of feminist thought. Well not introduced exactly, more like pummeled. Here is what I have learned. I have something called. “white male privilege”. Essentially, whether I consciously or subconsciously acknowledge that privilege, it doesn’t matter. I have it and I need to “own it”. I’m pretty sure that means I have to fess up to it and wear it like a scarlet letter (although a white penis will do just fine). Believe me! I understand the importance of the concept. The dried blood tracking from my ears is proof positive of the difficulties and hard work it took me to reach that understanding. But that’s as far as the feminists have taken me. I’m afraid to tell them but it’s like a false crescendo. It can’t be the end of the symphony. Okay, so I “own” white male privilege. What next? There must be more. Am I supposed to give it up someday? Is it like owning an unregistered gun? Will there be a turn-in-your-white-male-privilege amnesty day? I’d be more than happy to if I just knew when, where and to whom? Or come to think of it, maybe not. What takes its place? Or worse, who gets it next? Gee, maybe I should take advantage of it more consciously while I still have it. Anyway, in the meantime, as I meander aimlessly, I’m going to refrain from saying stuff like, “Hey that Barbara Boxer, she sure has some pouch of brass nuggets on her.” I will also try to be more cognizant of this privilege I have and renounce it at every turn. It’s all I can do until I get further instructions. You know, I can’t help but think if reincarnation happens, I might want to come back as an earthworm. They have both the male and female sex organs. When they mate they impregnate each other. Everything is “even up”. And the result is that they are a pretty happy bunch. You don’t hear about earthworms having male/female issues. Okay so they have other issues—fish hooks being a big one. 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As a Horror writer I've been often and pointedly been asked why I write this stuff. It's not ever said directly, but it's always there: Is there something wrong with you? In my own defense, quite a few people enjoy reading this same stuff and even more get a thrill out of watching it on the big screen. Just to hazard a guess, I'd say most people have in their life read a horror book or seen a horror movie. The question then becomes: What's wrong with us? My first occasions to hear horror stories was as a child in church. I was told that there was a man in a red suit and horns who carried a pitchfork and watched everything I did and wanted to send me to the worst, most horrible place ever if I did bad things. Worse than this, I was told that there was something called 'original sin' and just by being born I was on God's crap list and if I didn't repent for things I'd never done, the man in the red suit would still get me. It didn't seem quite fair to me that my little three year old wrong-doings could earn me the same trip to Hell that someone like Hitler got. I was scared constantly. And that was the point of those stories, to scare little boys into behaving as their parents wanted them to. Fairy tales have the same theme: Obey your parents, or bad things will happen. I can't swear that I remember all of my fairy tales, but I do remember as a child being - probably - unreasonably worried about being eaten. For the time, being eaten seemed about the worst thing that could happen to me and I looked warily at strangers trying to evaluate in my mind whether they would try and eat me. Fortunately, there were very few cannibals in Wisconsin at that time. Jeffrey Dahmer was one, but for the life of me, I can't think of any other Wisconsin cannibals. Oh, wait. Ed Gein - but that's it. Parents frightening their kids is one thing, but why do people want to scare themselves? Did you ever wonder why you paid good money at the bookstore and at the movies for this service that your parents would happily provide you for free? Well, horror stories are about fear, but it's not just about making yourself scared - that alone is no fun. Horror stories are about conquering your fear, and the way they do that is symbolically by creating a monster that represents a fear and by having that monster defeated. Thus it helps you to overcome your subconscious fear/Monster by identifying with the destruction of the one in the story. Works out pretty neat, huh? Here's how it plays out in a few familiar scenarios. Frankenstein, by Mary Shelley, was thought to the first real science fiction book, although it really is a horror story. In the story Victor Frankenstein discovers the secret of life - itself! As an experiment he creates for himself a man sewn together from cadavers and then embues it with life, and then seeing what an awful looking creature he's created, he abandons it. He does this because it looks so hideous, though for the life of me, I can't figure out why he had to make the thing out of several icky corpses instead of just finding one beautiful one and giving that one life. Anyways, the monster runs away and then comes back to haunt him and he has to destroy it. The explanation for Frankenstein is that the monster represents science and the Victorian fear that science and progress had gone too far. Science, once the obedient servant of mankind, had, like Frankenstein's monster, broken free and turned against its master - us. A hundred or years later this same theme is echoed in the movie The Terminator, only this time the science that breaks free is computer science. Computers, our formerly docile servant, turn against us and band together to become one giant warlike mind which for some reason or other decides that all humans must perish throughout time. I guess we had it coming to us. Vampires, another popular monster, have represented the once prevalent infectious disease that used to regularly wipe out giant swathes of human population. In modern times, Vampires have been reinterpeted to be kind of sexy, that is, they represent the dark sexual impulses people have inside themselves that they also think may destroy them. Vampire stories, then, become our victory over our dark, forbidden desires. Which are represented by those sexy, sexy vampires. Sex is a constant theme in the slasher movies. The Scream movies brilliantly satirize this by having the teen-agers in the movie aware of the conventions of the genre they are living through, yet helpless to change them as those conventions become their fates. In the slasher movies young girls fear of their own sexual maturity is confronted symbolically by the slasher who represents teen-age boys through the menace of wielding the very Freudian penis/knife. You'll notice that the heroine that inevitably prevails in these movies is the virgin who never succombs to the temptation of sex and not coincidentally, does not succomb to the slasher, either. My favorite monsters are the ones from the Japanese monster movies, Godzilla, Mothra, Rodan and, of course, Monster Zero. The reason I love these monsters is that they are political monsters. Think about it: Godzilla is a giant, super-powerful radioactive monster who comes from over the sea who is created by radioactivity and then attacks Japan with that same radioactivity. Sound familiar? (Hint: It's America). All these monsters from overseas are constantly attacking Japan and being beaten up by the cohesion of the Japanese people. 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Testosterone is an androgenic hormone produced chiefly by the testes. It is responsible for the growth of secondary sex characteristics in men. A small amount of testosterone is present in the body of women also. The deficiency or excessive amounts of this hormone causes many side effects in the human body. Many debates have been conducted among scientists and healthcare professionals on the side effects of testosterone. Almost all unwanted side effects of testosterone are caused the androgenic properties of the hormone. The usage of synthetic testosterone helps people with HIV-related wasting gain weight. A man with testosterone deficiency experiences decreased sex drive, moodiness, and fatigue. Testicular function deceases with age. This phenomenon is usually found in men after the age of thirty. The supplementation of the hormone sometimes tends to slow down the body?s natural ability to produce testosterone. This is the major side effect found in men. Vomiting, nausea, swelling of the arms and legs, yellowing of the skin and eyes, and prolonged and painful erections are the early signs of serious side effects. Using excess amount of artificial testosterone causes serious allergic reactions such difficulty in breathing, swelling of the tongue, lips, or face. In women, hoarseness, male-pattern baldness, deepening of voice, excessive hair growth, and menstrual irregularities are the main side effects. Prolonged use of testosterone in higher doses may result in shrinking of testicles, gynecomastia (breast growth in men), decreased or increased sex drive, and a number of less serious side effects such as acne, decreased sperm production in men, clitoral enlargement, male pattern baldness, and water retention. Liver damage and cancer are the other serious side effects caused by testosterone.